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Benfotiamine increases tissue amounts of thiamine diphosphate, consequently growing transketolase activity and producing a significant decrease in glucose metabolites and precursors to ages.

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To investigate the effect of benfotiamine on urinary excretion of albumin and the tubular damage marker kidney injury molecule-1 kim-1 in patients with type 2 diabetes and nephropathy.

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Benfotiamine prevents increased -amyloid production in hek cells induced by high glucose.

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